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The activities involved in receiving an SCH burst are shown in Figure 17.7. The tasks involved are: (i) Programming the hardware with the frequency to receive and the time at which to start the receive. This is shown as task P. This task is triggered by the main layer 1 state machine when an SCH burst receive is required Reading blocks of 16 sample pairs from HW buffer to main memory buffer. This task is not shown since it is too frequent. After 11 blocks have been received, task (iii) is triggered. 176 sample pairs are used instead of the usual 160 since there is generally more uncertainty about the precise timing of a sync burst, since the only timing information may have come from the previous FCCH receive. Non-FCCH bursts contain a known sequence of bits (the midamble) which can be used to estimate the burst timing accurately Pass the 176 received sample pairs through the sync burst equalizer (EQ). This calculates the precise timing of the burst and demodulates it Pass the demodulated burst through the channel decoder (CD). This corrects any errors in the received burst. This task is triggered by the end of the EQ task Reprogram the hardware to stop the receive occurring again. This is shown as task Q. It is triggered by the per-frame interrupt following the receive period.

as the routes from the sensors to the sinks Time is divided into rounds At the beginning of each round, information on the nodes residual energy is centrally gathered and the ILP problem is solved for nding the new locations for the sink that minimize the maximum energy consumption spent at the nodes during that round Minimizing the energy consumption results in increased network longevity No constraints are enforced on the sink movements, and there is no relation between the number of the sinks and their position in subsequent rounds: The model is solved from scratch every time The problem of network lifetime maximization through controlled sink mobility is explicitly addressed in reference 54 for networks with a single sink Sink locations and sink sojourn times at those locations are determined that maximize the network lifetime via a new LP formulation of the problem.

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53 A series of studies were then carried out by Quenelle and co-workers54 to determine the comparative ef cacy of parenteral CDV with oral HDP-CDV and ODE-CDV, on CV and VV infections of mice Since we have reported previously that CDV is highly active in these models when given as single or multiple doses either prior to or after infection, similar studies were carried out with these analogues In addition, we also determined the effect of oral treatment with HDP-CDV or ODE-CDV on the replication of CV or VV in important target organs In initial experiments, HDP-CDV or ODE-CDV were administered once daily for 5 consecutive days by oral gavage to CV- or VV-infected mice beginning 24, 48, or 72 h post viral inoculation CDV was given ip at similar doses and times of initiation of therapy At the 6.

(ii)

Maximizing the network lifetime is maximizing the sum of sojourn times of the sink at the visited locations (in this case they coincide with the sensor locations) The experiments in reference 54 refer to scenarios where n = L2 nodes are arranged in a L L grid The sink has no limitation on the time tk 0 it can spend at sensor k and can move from any location to any other location in the network The network lifetime is improved by a factor of 5 This happens when the sink sojourns at the nodes located at the four corner areas and in the central area of the grid Papadimitriou and Georgiadis [55] present another centralized solution for the problem of maximizing network lifetime by combining the model presented in reference 54 and the LP formulation for maximum lifetime routing described in reference 57.

7 mg/kg dose, no toxicity was observed and each compound signi cantly reduced nal mortality in CV-infected mice (p 001) at one or more times of initiation of therapy (Table 149) In mice inoculated with VV, both HDP-CDV and ODE-CDV signi cantly reduced mortality rates when treatment was initiated as late as 48 h post viral inoculation Treatment with CDV also resulted in signi cant protection from mortality and at most times of initiation of treatment In mice infected with ectromelia virus (mousepox), similar results were obtained to those seen above for VV and CV48 At 10 mg/kg both analogues gave complete protection, whereas ODE-CDV was more effective at lower dosages than HDP-CDV We have reported previously that CDV can protect mice infected with CV or VV when given as early as 5 days prior to infection.

A constant of the model in reference 54 is here turned into a variable so that the resulting model presented solves both (a) the problem of determining the sink sojourn times at the given sites and (b) the routing of the packets to the current position of the sink This routing-dependent solution achieves improvements with respect to the lifetime values of reference 54 that are twofold Lifetime maximization as a min max problem is the idea explored by Luo and Hubaux [56] The authors consider together sink mobility and data routing A load balancing solution is presented that, while keeping the sink moving along the external perimeter of the network, achieves lifetimes 500% higher than when the sink stays still in the center of the network.

(iii)

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